NEUROVASCULAR

COMPRESSION

 

 

NEUROVASCULAR COMPRESSION

 

 

 

A neurovascular compression is described as a prominent pathological contact between cranial nerves and vessels. This contact builds up in the so-called root entry zone of a cranial nerve, where we find the transition of central to peripheral myelin resulting in less myelination of nerve fibres (axon). This region varies in length depending on the different cranial nerves and is located close to the brainstem. This results in typical hyperactive dysfunction of the respective cranial nerve. The trigeminal neuralgia, the spasmus hemifaciei (hemifacial spasm) and the glossopharyngeal neuralgia are typical clinical entities associated with neurovascular compression as the predominant cause.


There is evidence that a number of other diseases, such as arterial hypertension and certain forms of vertigo (dizziness) and tinnitus are closely related to neurovascular compression. Microvascular decompression is the established causal surgical method for the treatment of syndromes caused by neurovascular compression. With high resolution magnet resonance imaging scanners and subsequent application of image processing neurovascular compression can be visualized in a 3D representation.

 

 

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INTRODUCTION

CONSULTATION

NEUROVASCULAR COMPRESSION

ENTITIES

TRIGEMINAL NEURALGIA

HEMIFACIAL SPASM

GLOSSOPHARYNGEAL NEURALGIA

ARTERIAL HYPERTENSION

MICROVASCULAR DECOMPRESSION

THERMOCOAGULATION

STEREOTACTIC RADIATION

RESEARCH

PUBLICATIONS

NEWS, LINKS

CONTACT, APPROACH

IMPRESSUM

 

             

 

 

Principle  of neurovascular compression

 

 

 

 

Models of pathophysiology of the neurovascular compression

 

 

 

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R. NARAGHI